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Researchers Identify Potential New Approach for Improving Quality of Life  for ALS Patients - Neuroscience News
Researchers Identify Potential New Approach for Improving Quality of Life for ALS Patients - Neuroscience News

MicroRNA-206 Delays ALS Progression and Promotes Regeneration of  Neuromuscular Synapses in Mice | Science
MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice | Science

Innate immune adaptor TRIF confers neuroprotection in ALS mice by  eliminating abnormal glial cells | Asia Research News
Innate immune adaptor TRIF confers neuroprotection in ALS mice by eliminating abnormal glial cells | Asia Research News

Motor neuron disease, TDP-43 pathology, and memory deficits in mice  expressing ALS–FTD-linked UBQLN2 mutations | PNAS
Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS–FTD-linked UBQLN2 mutations | PNAS

IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A  Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic  Lateral Sclerosis
IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic Lateral Sclerosis

Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic  lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular  atrophy protein to nuclear Cajal bodies. | Semantic Scholar
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar

Modelling amyotrophic lateral sclerosis in rodents | Nature Reviews  Neuroscience
Modelling amyotrophic lateral sclerosis in rodents | Nature Reviews Neuroscience

Sex-Specific Differences in Motor-Unit Remodeling in a Mouse Model of ALS |  eNeuro
Sex-Specific Differences in Motor-Unit Remodeling in a Mouse Model of ALS | eNeuro

JCI - Membralin deficiency dysregulates astrocytic glutamate homeostasis,  leading to ALS-like impairment
JCI - Membralin deficiency dysregulates astrocytic glutamate homeostasis, leading to ALS-like impairment

Tempol improves neuroinflammation and delays motor dysfunction in a mouse  model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text

Stage-dependent remodeling of projections to motor cortex in ALS mouse model  revealed by a new variant retrograde-AAV9 | eLife
Stage-dependent remodeling of projections to motor cortex in ALS mouse model revealed by a new variant retrograde-AAV9 | eLife

Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a  Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis
Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis

Drug target validation in mouse models of ALS. (A) Comparison between... |  Download Scientific Diagram
Drug target validation in mouse models of ALS. (A) Comparison between... | Download Scientific Diagram

A major step towards the cure of sporadic ALS | The University of Tokyo
A major step towards the cure of sporadic ALS | The University of Tokyo

AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a  therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods &  Clinical Development
AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods & Clinical Development

Humanising mice to enable modelling of neurodegenerative diseases | UCL  Queen Square Institute of Neurology - UCL – University College London
Humanising mice to enable modelling of neurodegenerative diseases | UCL Queen Square Institute of Neurology - UCL – University College London

Working with ALS Mice: Guidelines for Preclinical Testing and Colony  Management
Working with ALS Mice: Guidelines for Preclinical Testing and Colony Management

Loss of Stathmin-2, a hallmark of TDP-43-associated ALS, causes motor  neuropathy - ScienceDirect
Loss of Stathmin-2, a hallmark of TDP-43-associated ALS, causes motor neuropathy - ScienceDirect

Amyotrophic Lateral Sclerosis: Marking the differences in motoneurons |  eLife
Amyotrophic Lateral Sclerosis: Marking the differences in motoneurons | eLife

TDP-43 mutant transgenic mice develop features of ALS and frontotemporal  lobar degeneration | PNAS
TDP-43 mutant transgenic mice develop features of ALS and frontotemporal lobar degeneration | PNAS

A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores  Function in an ALS Mouse Model - ScienceDirect
A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect

Gut microbes tune inflammation and lifespan in a mouse model of amyotrophic  lateral sclerosis
Gut microbes tune inflammation and lifespan in a mouse model of amyotrophic lateral sclerosis

Amyotrophic lateral sclerosis (ALS):SOD1 mouse helps researchers identify  immune component
Amyotrophic lateral sclerosis (ALS):SOD1 mouse helps researchers identify immune component

Opinion: more mouse models and more translation needed for ALS | Molecular  Neurodegeneration | Full Text
Opinion: more mouse models and more translation needed for ALS | Molecular Neurodegeneration | Full Text

Neuromuscular Junction Dysfunction in Amyotrophic Lateral Sclerosis |  SpringerLink
Neuromuscular Junction Dysfunction in Amyotrophic Lateral Sclerosis | SpringerLink

Rodent Amyotrophic Lateral Sclerosis (ALS) Model - Creative Biolabs
Rodent Amyotrophic Lateral Sclerosis (ALS) Model - Creative Biolabs

November 2015 Pathomechanistic model mice of sporadic ALS: Conditional  ADAR2 knockout mice AR2 | Experimental Animal Division (RIKEN BRC)
November 2015 Pathomechanistic model mice of sporadic ALS: Conditional ADAR2 knockout mice AR2 | Experimental Animal Division (RIKEN BRC)