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Researchers Identify Potential New Approach for Improving Quality of Life for ALS Patients - Neuroscience News
MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice | Science
Innate immune adaptor TRIF confers neuroprotection in ALS mice by eliminating abnormal glial cells | Asia Research News
Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS–FTD-linked UBQLN2 mutations | PNAS
IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic Lateral Sclerosis
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar
Modelling amyotrophic lateral sclerosis in rodents | Nature Reviews Neuroscience
Sex-Specific Differences in Motor-Unit Remodeling in a Mouse Model of ALS | eNeuro
JCI - Membralin deficiency dysregulates astrocytic glutamate homeostasis, leading to ALS-like impairment
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
Stage-dependent remodeling of projections to motor cortex in ALS mouse model revealed by a new variant retrograde-AAV9 | eLife
Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis
Drug target validation in mouse models of ALS. (A) Comparison between... | Download Scientific Diagram
A major step towards the cure of sporadic ALS | The University of Tokyo
AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods & Clinical Development
Humanising mice to enable modelling of neurodegenerative diseases | UCL Queen Square Institute of Neurology - UCL – University College London
Working with ALS Mice: Guidelines for Preclinical Testing and Colony Management
Loss of Stathmin-2, a hallmark of TDP-43-associated ALS, causes motor neuropathy - ScienceDirect
Amyotrophic Lateral Sclerosis: Marking the differences in motoneurons | eLife
TDP-43 mutant transgenic mice develop features of ALS and frontotemporal lobar degeneration | PNAS
A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect
Gut microbes tune inflammation and lifespan in a mouse model of amyotrophic lateral sclerosis